Why Get Your Genetic Background Tested?
Genetic Ancestry Report
Admixture analysis (more properly known as biogeographical ancestry analysis) is a method of inferring
someone’s geographical origins based on an analysis of their genetic ancestry. An admixture analysis is one
of the components of an autosomal DNA test. Admixture calculations provide genetic ancestry analysis to
individuals tested for singlenucleotide polymorphism (SNP) data. Admixture analysis usually builds ancestral
components also called clusters by comparing a dataset of samples. Both the used datasets (regional, continental,
worldwide) and the ancestral components (number, age) are very diverse depending on the used
setup and analysis method. A new sample (not used in the dataset) is normally compared to the ancestral
components by the calculation of the percentages. Additional tools allow also the prediction of ancestral
How The Test Works
Order Kit Online
Create an account online and purchase your test. We will ship collection kit directly to your home.
Provide sample using the collection kit, then ship back using the prepaid packaging.
Get your Results
We will process and analyze your genes. Then, you can begin to explore your genetic ancestry!
Your Detailed Results Include
Discover your ethnic background using your genes.
Have you always been told you look Irish but you have been raised Italian? Curious to see find out where you come from? Begin to trace your ancestry and discover more about yourself! DNA can be the first step in self-discovery.
ALDH2, OPRM1, DRD2, CYP1A2, MCM6, HLA-DQA1, FTO, MGAM, TAS2R38, ANKK1, TAS2R38, FTO, APOA2, LOC100507686, HLA-DRA, FTO, ADRB2, ADRB3, APOA2, FABP2, PPARG, APOA5, ADR83, ADRB2, PPARG, FABP2, ADRB2
Alcohol Reaction, Coffee (Caffeine) Metabolism, Lactose Intolerance, Gluten intolerance (Celiac disease), Sweet Sensitivity, sweet tooth, bitter taste, Eating Disinhibition, Food Desire, Hunger, Snacking, Satiety – Feeling Full, Sodium Restricted Diet, Saturated fat to Obesity, Folate Diet, Peanut Allergy
- This may be why that popular diet didn’t work for you
- Kilpelainen, T. O., Qi, L., Brage, S., Sharp, S. J., Sonestedt, E., Demerath, E., Loos, R. J. (2011). Physical activity
attenuates the influence of FTO variants on obesity risk: a meta-analysis of 218,166 adults and 19,268 children. PLoS
Medicine, 8 (11), e1001116. doi:10.1371/journal.pmed.1001116
- Li, S., Zhao, J. H., Luan, J., Ekelund, U., Luben, R. N., Khaw, K. T., . . . Loos, R. J. (2010). Physical activity attenuates
the genetic predisposition to obesity in 20,000 men and women from EPIC-Norfolk prospective population study. PLoS
Medicine, 7(8), e1000332. doi:10.1371/journal.pmed.1000332
- Vimaleswaran, K. S., Li, S., Zhao, J. H., Luan, J., Bingham, S. A., Khaw, K. T., . . . Loos, R. J. (2009). Physical activity
attenuates the body mass index-increasing influence of genetic variation in the FTO gene. The American Journal of
Clinical Nutrition, 90(2), 425-428. doi:10.3945/ajcn.2009.27652
- Duell, E. J., Lujan-Barroso, L., Llivina, C., Munoz, X., Jenab, M., Boutron-Ruault, M. C., . . . Gonzalez, C. A. (2013).
Vitamin C transporter gene (SLC23A1 and SLC23A2) polymorphisms, plasma vitamin C levels, and gastric cancer risk in
the EPIC cohort. Genes & Nutrition, 8(6), 549-560. doi:10.1007/s12263-013-0346-6
- Kapur, K., Johnson, T., Beckmann, N. D., Sehmi, J., Tanaka, T., Kutalik, Z., . . . Bergmann, S. (2010). Genome-wide
metaanalysis for serum calcium identifies significantly associated SNPs near the calcium-sensing receptor (CASR) gene.
PLoS Genetics, 6(7), e1001035. doi:10.1371/journal.pgen.1001035
- Kobylecki, C. J., Afzal, S., Davey Smith, G., & Nordestgaard, B. G. (2015). Genetically high plasma vitamin C, intake of
fruit and vegetables, and risk of ischemic heart disease and all-cause mortality: a mendelian randomization study. The
American Journal of Clinical Nutrition, 101(6), 1135-1143. doi:10.3945/ajcn.114.104497
- Lin, X., Lu, D., Gao, Y., Tao, S., Yang, X., Feng, J., . . . Sun, J. (2012). Genome-wide association study identifies novel
loci associated with serum level of vitamin B12 in Chinese men. Human Molecular Genetics, 21(11), 2610-2617.
- 8Lu, L., Sheng, H., Li, H., Gan, W., Liu, C., Zhu, J., . . . Lin, X. (2012). Associations between common variants in GC and
DHCR7/NADSYN1 and vitamin D concentration in Chinese Hans. Human Genetics, 131(3), 505-512. doi:10.1007/s00439-
- Lu, Y., Vaarhorst, A., Merry, A. H. H., Dolle, M. E. T., Hovenier, R., Imholz, S., . . . Feskens, E. J. M. (2012). Markers of
endogenous desaturase activity and risk of coronary heart disease in the CAREMA cohort study. PLoS One [electronic
resource], 7(7), e41681. doi:10.1371/journal.pone.0041681
- Mondul, A. M., Yu, K., Wheeler, W., Zhang, H., Weinstein, S. J., Major, J. M., . . . Albanes, D. (2011). Genome-wide
association study of circulating retinol levels. Human Molecular Genetics, 20(23), 4724-4731
- Frayling et al. (2007). “A common variant in the FTO gene is associated with body mass index and predisposes to
childhood and adult obesity.” Science 316(5826):889-94. ·
12. Dina et al. (2007). “Vari ation in FTO contributes to childhood obesity and severe adult obesity.” Nat Genet 39(6):724-
- Scuteri et al. (2007). “Genome-wide association scan shows genetic variants in the FTO gene are associated with
obesityrelated traits.” PLoS Genet 3(7):e115
- Roth SM1, Walsh S, Liu D, Metter EJ, Ferrucci L, Hurley BF.(2008) The ACTN3 R577X nonsense allele is underrepresented
in elite-level strength athletes Eur J Hum Genet. 2008 Mar;16(3):391-4
- Wichmann HE. Et.al (2009) Meta-analysis of the INSIG2 association with obesity including 74,345 individuals: does
heterogeneity of estimates relate to study design PLoS Genet. 2009 Oct;5(10):e1000694. Doi 1
- Rogozkin VA.et.a. (2009) The combined impact of metabolic gene polymorphisms on elite endurance athlete status
and related phenotypes0.1371 Hum Genet. 2009 Dec;126(6):751-61. doi: 10.1007/s00439-009-0728-4
- Franks PW. et.al. (2008) The Gly482Ser genotype at the PPARGC1A gene and elevated blood pressure: a metaanalysis
involving 13,949 individuals. J Appl Physiol (1985). 2008 Oct;105(4):1352-8. doi: 10.1152/
Diet & Exercise Match Powered by Diagnomics
- Johnson JA. Et.al. (2008) Adrenergic gene polymorphisms and cardiovascular risk in the NHLBI-sponsored Women’s
Ischemia Syndrome Evaluation J Transl Med. 2008 Mar 10;6:11. doi: 10.1186/1479-5876-6-11
- Taylor J1, Sun YV, Chu J, Mosley TH, Kardia SL.(2012) Interactions between metallopeptidase 3 polymorphism
rs679620 and BMI in predicting blood pressure in African-American women with hypertension J Hypertens. 2008
Dec;26(12):2312-8. doi: 10.1097/HJH.0b013e3283110402
- Beilby JP.et.al. (2008) Investigating the association between K198N coding polymorphism in EDN1 and hypertension,
lipoprotein levels, the metabolic syndrome and cardiovascular disease. Hum Genet. 2008 Apr;123(3):307-13. doi: 10.100
- Gallicchio L1, Kalesan B, Huang HY, Strickland P, Hoffman SC, Helzlsouer KJ.(2008) Genetic polymorphisms of
peroxisome proliferator-activated receptors and the risk of cardiovascular morbidity and mortality in a community-based
cohort in washington county, Maryland. PPAR Res. 2008;2008:276581
- Gonzalez-Santos P.et.al (2010) Additive effects of LPL, APOA5 and APOE variant combinations on triglyceride levels
and hypertriglyceridemia: results of the ICARIA genetic sub-study BMC Med Genet. 2010 Apr 29;11:66
- Verhoeven AJ et.al. (2008) High HDL cholesterol does not protect against coronary artery disease when associated
with combined cholesteryl ester transfer protein and hepatic lipase gene variants Atherosclerosis. 2008 Sep;200(1):161-7
Frequently Asked Questions
Who will see my results?
Your results will be available to you only. You can choose to share it with your friends and family. This information can connect your biology to others areas of you life from products to wellness plans.
How accurate are my results?
This analysis is based on high throughput sequencing or genotyping data from a testing sample. The report does not provide any medical advice, diagnosis or treatment. These results are for information purposes and subject to change. You should consult your physician if you have questions regarding any medical condition or treatment. The results and analysis presented in this report have not been clinically validated. nor cleared or approved by the FDA or similar government institutions.
This test was developed and its performance characteristics determined by the lab. FDA approval is not currently required for use of this test. Lab validation was done as required by the Clinical Laboratory Improvement Amendments of 1988 (CLIA).